Post-doc to study CED-9 IP3R interaction's influence on Ca2+ signaling

Our lab has a postdoctoral position opening in December to study how intrinsic cell death and particularly Bcl2 proteins regulate calcium signaling through interacting with the InsP3 receptor. The general approach includes using CRISPR to disrupt contact sites between these two proteins and biosensors to integrate localized calcium signaling at the mito-ER interface with the behavioral effects of loss-of-interaction. Candidates can send their CV to Information on Rochester and NehrkeLab can be found at